Alzheimer publications Delacourte

Publications 2006 24/03/09

Buee L. and Delacourte A. (2006) [Tauopathy and Alzheimer disease: a full degenerating process]. Psychol Neuropsychiatr Vieil 4, 261-273.

DELACOURTE A. De la physiopathologie au traitement de la maladie d’Alzheimer. Rev Neurol (Paris) 2006 ; 162 : 10, 909-912 [From physiopathology to treatment of Alzheimer's disease]

Delacourte A. (2006) The natural and molecular history of Alzheimer's disease. J Alzheimers Dis 9, 187-194.

ALZ100
The natural and molecular history of Alzheimer's disease

Delacourte A. (2006) The natural and molecular history of Alzheimer's disease: tau is part of the story. Research and Perspectives in Alzheimer's disease, 325-332. Tubingen.

Delacourte A. Amyloid Precursor Protein Neurotrophic Properties As a Target to Cure Alzheimer’s Disease. European Neurological Disease 2006 2006;2006:29,30.

EMC Alzheimer: le texte

Deramecourt V, Bombois S, Maurage CA, Ghestem A, Drobecq H, Vanmechelen E, Lebert F, Pasquier F, Delacourte A.

Biochemical staging of synucleinopathy and amyloid deposition in dementia with lewy bodies.

J Neuropathol Exp Neurol. 2006 Mar;65(3):278-288

Hamdane M, Dourlen P, Bretteville A, Sambo AV, Ferreira S, Ando K, Kerdraon O, Begard S, Geay L, Lippens G, Sergeant N, Delacourte A, Maurage CA, Galas MC, Buee L.
Pin1 allows for differential Tau dephosphorylation in neuronal cells.
Mol Cell Neurosci. 2006 May 9; [Epub ahead of print]
PMID: 16697218

Husson M., Enderlin V., Delacourte A., Ghenimi N., Alfos S., Pallet V. and Higueret P. (2006) Retinoic acid normalizes nuclear receptor mediated hypo-expression of proteins involved in beta-amyloid deposits in the cerebral cortex of vitamin A deprived rats. Neurobiol Dis 23, 1-10.

Husson M, Enderlin V, Delacourte A, Ghenimi N, Alfos S, Pallet V, Higueret P

Leroy O., Dhaenens C. M., Schraen-Maschke S., Belarbi K., Delacourte A., Andreadis A., Sablonniere B., Buee L., Sergeant N. and Caillet-Boudin M. L. (2006) ETR-3 represses Tau exons 2/3 inclusion, a splicing event abnormally enhanced in myotonic dystrophy type I. J Neurosci Res 84, 852-859.

Hamdane M., Dourlen P., Bretteville A., Sambo A. V., Ferreira S., Ando K., Kerdraon O., Begard S., Geay L., Lippens G., Sergeant N., Delacourte A., Maurage C. A., Galas M. C. and Buee L. (2006) Pin1 allows for differential Tau dephosphorylation in neuronal cells. Mol Cell Neurosci 32, 155-160.